What was it all about?
Persimmon dieback is an increasing problem in Australian orchards, reducing tree health, productivity and, in severe cases, causing tree death. This project improved industry understanding of what causes dieback and how it can be better managed.
The research confirmed that dieback in Australian persimmons is not caused by a single pathogen. Instead, it involves a group of different fungi, some of which are more aggressive than others. This finding explains why symptoms can vary between orchards and why management has been challenging.
To build this understanding, researchers surveyed orchards across all major persimmon‑growing regions and collected samples from affected trees. Fungi linked to dieback were identified using laboratory and DNA-based testing, then tested to confirm which ones caused disease. The work also examined how infection occurs, how disease develops inside trees, whether different cultivars respond differently, and whether the fungi can infect other crops or come from nursery planting material. Fungicides and biological products were tested to identify options that may help reduce disease risk.
The project addressed a critical knowledge gap that made dieback difficult to diagnose and manage. It showed that infection often starts through fresh wounds, such as pruning cuts, and that disease is usually well established before symptoms are visible.
The findings highlight that preventing infection is far more effective than trying to cure it later. Several fungicides and biological products showed promise when applied preventatively, giving growers clearer, evidence-based options to protect trees and support long‑term orchard productivity.